Journal of Preventive Medicine and Public Health

Young Ju Kim

6 Articles

Body Weight at Birth and at Age Three and Respiratory Illness in Preschool Children.: Yoolwon Jeong, Kyunghee Jung-Choi, Jin Hwa Lee, Hwa Young Lee, Eun Ae Park, Young Ju Kim, Eunhee Ha, Se Young Oh, Hyesook Park; J Prev Med Public Health. 2010;43(5):369-376.; DOI: https://doi.org/10.3961/jpmph.2010.43.5.369

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OBJECTIVES
The purpose of this study was to examine the associations of current body weight and body mass index (BMI) at age three and birth weight in developing chronic respiratory illness in childhood and identify possible interaction underlying its mechanism. METHODS: The study was carried out with 422 children who were enrolled in a hospital-based birth cohort. Birth related anthropometric data were collected at birth. At age 3 years, the presence of respiratory symptoms was evaluated by using the Korean version of core questionnaire for wheezing and asthma from the International Study of Asthma and Allergies in Childhood (ISAAC). Physical examination was carried out to measure the child's weight and height. RESULTS: Children in the lowest birth weight tertile (aOR = 3.97, 95% CI = 0.94-16.68) or highest BMI tertile (aOR = 3.68, 95% CI = 1.24-10.95) at three years of age were at an increased risk of chronic respiratory illness. Children who were initially in the lowest birth weight tertile but now belong in the highest weight tertile had higher risk of chronic respiratory illness compared to those who had remained in the middle tertile (OR=16.35, 95% CI=1.66-160.57). CONCLUSIONS: Children with lower birth weight or higher BMI were at an increased risk of chronic respiratory illness. In addition, children who were initially in the lowest birth weight tertile but are now in the highest weight tertile had higher risk of chronic respiratory illness compared to those who remained in the middle tertile.

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Makiko Nanishi, Michimasa Fujiogi, Michelle Stevenson, Liming Liang, Ying Shelly Qi, Yoshihiko Raita, Kohei Hasegawa, Carlos A. Camargo
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Lifetime overweight and adult asthma: 1978/1979 Ribeirão Preto Birth Cohort, São Paulo, Brazil
Luana Lopes Padilha, Cecilia Claudia Costa Ribeiro, Joelma Ximenes Prado Teixeira Nascimento, Vanda Maria Ferreira Simões, Fernanda Pino Vitti, Viviane Cunha Cardoso, Elcio Oliveira Vianna, Marco Antônio Barbieri, Antônio Augusto Moura da Silva, Heloísa B
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Yue Zhang, Zhanghua Chen, Kiros Berhane, Robert Urman, Vaia Lida Chatzi, Carrie Breton, Frank D Gilliland
American Journal of Epidemiology.2020; 189(6): 583. CrossRef
Changes in body mass index during childhood and risk of various asthma phenotypes: a retrospective analysis
Julie Chastang, Nour Baiz, Laure Parnet, Jean Sébastien Cadwallader, Frédéric De Blay, Denis Caillaud, Denis André Charpin, John Dwyer, François Lavaud, Chantal Raherison, Gladys Ibanez, Isabella Annesi‐Maesano
Pediatric Allergy and Immunology.2017; 28(3): 273. CrossRef
Association between obesity and asthma – epidemiology, pathophysiology and clinical profile
Magdalena Muc, Anabela Mota-Pinto, Cristina Padez
Nutrition Research Reviews.2016; 29(2): 194. CrossRef
Excess weight in preschool children with a history of severe bronchiolitis is associated with asthma
Sari Törmänen, Eero Lauhkonen, Antti Saari, Petri Koponen, Matti Korppi, Kirsi Nuolivirta
Pediatric Pulmonology.2015; 50(5): 424. CrossRef
Birth weight and childhood wheezing disorders: a systematic review and meta-analysis
Teumzghi F Mebrahtu, Richard G Feltbower, Darren C Greenwood, Roger C Parslow
Journal of Epidemiology and Community Health.2015; 69(5): 500. CrossRef
Mechanisms of obesity in asthma
Finn Rasmussen, Robert J. Hancox
Current Opinion in Allergy & Clinical Immunology.2014; 14(1): 35. CrossRef
Effects of prenatal community violence and ambient air pollution on childhood wheeze in an urban population
Yueh-Hsiu Mathilda Chiu, Brent A. Coull, Michelle J. Sternthal, Itai Kloog, Joel Schwartz, Sheldon Cohen, Rosalind J. Wright
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Early weight gain and the development of asthma and atopy in children
Gustavo F. Wandalsen, Herberto J. Chong-Neto, Fabíola S. de Souza, Dirceu Solé, Leonard B. Bacharier
Current Opinion in Allergy & Clinical Immunology.2014; 14(2): 126. CrossRef
Childhood Overweight/Obesity and Asthma: Is There a Link? A Systematic Review of Recent Epidemiologic Evidence
Constantina Papoutsakis, Kostas N. Priftis, Maria Drakouli, Stamatina Prifti, Eva Konstantaki, Maria Chondronikola, Georgios Antonogeorgos, Vasiliki Matziou
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Overview of Noncommunicable Diseases in Korean Children and Adolescents: Focus on Obesity and Its Effect on Metabolic Syndrome
Hye Ah Lee, Hyesook Park
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Fetal growth and risk of childhood asthma and allergic disease
S. G. Tedner, A. K. Örtqvist, C. Almqvist
Clinical & Experimental Allergy.2012; 42(10): 1430. CrossRef
Prenatal and Postnatal Maternal Stress and Wheeze in Urban Children
Yueh-Hsiu Mathilda Chiu, Brent A. Coull, Sheldon Cohen, Alana Wooley, Rosalind J. Wright
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Environmental Health Surveillance of Low Birth Weight in Seoul using Air Monitoring and Birth Data.: Ju Hee Seo, Eun Hee Ha, Ok Jin Kim, Byung Mi Kim, Hye Sook Park, Jong Han Leem, Yun Chul Hong, Young Ju Kim; J Prev Med Public Health. 2007;40(5):363-370.; DOI: https://doi.org/10.3961/jpmph.2007.40.5.363

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Abstract PDF

OBJECTIVES
The principal objective of this study was to determine the relationship between maternal exposure to air pollution and low birth weight and to propose a possible environmental health surveillance system for low birth weight. METHODS: We acquired air monitoring data for Seoul from the Ministry of Environment, the meteorological data from the Korean Meteorological Administration, the exposure assessments from the National Institute of Environmental Research, and the birth data from the Korean National Statistical Office between January 1, 2002 and December 31, 2003. The final birth data were limited to singletons within 37~44 weeks of gestational age. We defined the Low Birth Weight (LBW) group as infants with birth weights of less than 2500g and calculated the annual LBW rate by district. The air monitoring data were measured for CO, SO2, NO2, and PM10 concentrations at 27 monitoring stations in Seoul. We utilized two models to evaluate the effects of air pollution on low birth weight: the first was the relationship between the annual concentration of air pollution and low birth weight (LBW) by individual and district, and the second involved a GIS exposure model constructed by Arc View 3.1. RESULTS: LBW risk (by Gu, or district) was significantly increased to 1.113(95% CI=1.111~1.116) for CO, 1.004 (95% CI=1.003~1.005) for NO2, 1.202(95% CI=1.199~ 1.206) for SO2, and 1.077(95% CI=1.075~1.078) for PM10 with each interquartile range change. Personal LBW risk was significantly increased to 1.081(95% CI=1.002~1.166) for CO, 1.145(95% CI=1.036~1.267) for SO2, and 1.053(95% CI=1.002~1.108) for PM10 with each interquartile range change. Personal LBW risk was increased to 1.003(95% CI=0.954~1.055) for NO2, but this was not statistically significant. The air pollution concentrations predicted by GIS positively correlated with the numbers of low birth weights, particularly in highly polluted regions. CONCLUSIONS: Environmental health surveillance is a systemic, ongoing collection effort including the analysis of data correlated with environmentally-associated diseases and exposures. In addition, environmental health surveillance allows for a timely dissemination of information to those who require that information in order to take effective action. GIS modeling is crucially important for this purpose, and thus we attempted to develop a GIS-based environmental surveillance system for low birth weight.

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Spatiotemporal association of low birth weight with Cs-137 deposition at the prefecture level in Japan after the Fukushima nuclear power plant accidents: an analytical-ecologic epidemiological study
Hagen Scherb, Keiji Hayashi
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Current State of Research on the Risk of Morbidity and Mortality Associated with Air Pollution in Korea
Sanghyuk Bae, Ho-jang Kwon
Yonsei Medical Journal.2019; 60(3): 243. CrossRef
The Effect of Nitric Oxide Pollution on Oxidative Stress in Pregnant Women Living in Durban, South Africa
Samantha M. Anderson, Rajen N. Naidoo, Prithiksha Ramkaran, Alisa Phulukdaree, Sheena Muttoo, Kareshma Asharam, Anil A. Chuturgoon
Archives of Environmental Contamination and Toxicology.2018; 74(2): 228. CrossRef
HIV induced nitric oxide and lipid peroxidation, influences neonatal birthweight in a South African population
Samantha M. Anderson, Rajen N. Naidoo, Yashodani Pillay, Charlette Tiloke, Sheena Muttoo, Kareshma Asharam, Anil A. Chuturgoon
Environment International.2018; 121: 1. CrossRef
Outdoor Air Pollution, Preterm Birth, and Low Birth Weight: Analysis of the World Health Organization Global Survey on Maternal and Perinatal Health
Nancy L. Fleischer, Mario Merialdi, Aaron van Donkelaar, Felipe Vadillo-Ortega, Randall V. Martin, Ana Pilar Betran, João Paulo Souza
Environmental Health Perspectives.2014; 122(4): 425. CrossRef
Burden of Disease Due to Outdoor Air Pollution in Korea: Based on PM10
Hyun-Jin Kim, Seok-Jun Yoon, Hyeong-Su Kim, Kun-Sei Lee, Eun-Jung Kim, Min-Woo Jo, In-Hwan Oh
Korean Journal of Environmental Health Sciences.2011; 37(5): 387. CrossRef

Folate and Homocysteine Levels during Pregnancy affect DNA Methylation in Human Placenta.: Bo hyun Park, Young Ju Kim, Jong soon Park, Hwa young Lee, Eun hee Ha, Jung won Min, Hye sook Park; J Prev Med Public Health. 2005;38(4):437-442.

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Abstract PDF: OBJECTIVES
DNA methylation is one of the best characterized epigenetic mechanisms that play a regulatory role in genome programming and imprinting during embryogenesis. In this present study, we investigated the association between DNA methylation in the human placenta and the maternal folate and homocysteine concentrations on the Methylenetetrahydrofolatereductase (MTHFR) genetic polymorphism during pregnancy. METHODS: We investigated 107 pregnant women who visited Ewha Woman's University Hospital for prenatal care during their 24~28 weeks-period of gestation. During the second trimester, we measured the serum homocysteine and folate concentrations. The MTHFR 677 genetic polymorphism was determine by performing PCR-RFLP assay. The expression of DNA methylation in the human placentas was estimated by using immunohistochemistry method. RESULTS: Serum folate was negatively correlated with the serum homocysteine concentration for all the MTHFR genotypes. We found positive correlation between the folate concentrations and the DNA methylation in the human placenta (p< 0.05). An increasing concentration of homocysteine was associated with reduced DNA methylation in the human placenta. The coefficient value was -2.03 (-3.77, -0.29) on the regression model (p< 0.05). CONCLUSION: These findings suggest that the maternal folate and homocysteine levels along with the MTHFR 677 genetic polymorphism during pregnancy affect the DNA methylation in the human placenta.; Summary

Placental Superoxide Dismutase, Genetic Polymorphism, and Neonatal Birth Weight.: Yun Chul Hong, Kwan Hee Lee, Moon Whan Im, Young Ju Kim, Eun Hee Ha; J Prev Med Public Health. 2004;37(4):306-311. Published online November 30, 2004

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Abstract PDF: BACKGROUND
The roles of antioxidants in the placenta and genetic susceptibility to oxidant chemicals in relation to neonatal birth weight have not been elucidated. We determined whether the level of placental manganese superoxide dismutase (MnSOD) and its genetic polymorphism plays any role in oxidative stress and neonatal birth weight. METHODS: We measured placental MnSOD and determined MnSOD genetic polymorphism among 108 pregnant women who were hospitalized for delivery and their singleton live births in Korea. Main outcome measurements are maternal urinary malondialdehyde (MDA) and birth weight. RESULTS: Maternal urinary concentrations of MDA were significantly associated with neonatal birth weight (P=0.04). The enzyme level of placental MnSOD was also significantly associated with MDA concentration (P=0.04) and neonatal birth weight (P< 0.01). We observed dose-response relationships between placental MnSOD and maternal urinary MDA, and neonatal birth weight after adjusting for maternal weight, height, age, and neonatal sex. After controlling for covariates, MnSOD variant genotype increased maternal urinary MDA concentrations (P< 0.01) and reduced birth weight by 149 gm (P=0.08). CONCLUSIONS: This study demonstrates that the placental level of MnSOD during pregnancy significantly affects fetal growth by reducing oxidative stress, and that genetic polymorphism of MnSOD probably modulate the effects of oxidants on fetal growth.; Summary

Prenatal Exposure to PM10 and Preterm Birth between 1998 and 2000 in Seoul, Korea.: Eun Hee Ha, Bo Eun Lee, Hye Sook Park, Yun Sang Kim, Ho Kim, Young Ju Kim, Yun Chul Hong, Eun Ae Park; J Prev Med Public Health. 2004;37(4):300-305. Published online November 30, 2004

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Abstract PDF: OBJECTIVES
The exposure to particulate air pollution during the pregnancy has reported to result in adverse pregnancy outcome such as low birth weight, preterm birth, still birth, and intrauterine growth retardation (IUGR). We aim to assess whether prenatal exposure of particulate matter less than 10 (m in diameter (PM10) is associated with preterm birth in Seoul, South Korea. METHODS: We included 382, 100 women who delivered a singleton at 25-42 weeks of gestation between 1998 and 2000. We calculated the average PM10 exposures for each trimester period and month of pregnancy, from the first to the ninth months, based on the birth date and gestational age. We used three different models to evaluate the effect of air pollution on preterm birth; the logistic regression model, the generalized additive logistic regression model, and the proportional hazard model. RESULTS: The monthly analysis using logistic regression model suggested that the risks of preterm birth increase with PM10 exposure between the sixth and ninth months of pregnancy and the highest risk was observed in the seventh month (adjusted odds ratio=1.07, 95% CI=1.01- 1.14). We also found the similar results using generalized additive model. In the proportional hazard model, the adjusted odds ratio for preterm births due to PM10 exposure of third trimester was 1.04 (95% CI=0.96-1.13) and PM10 exposure between the seventh month and ninth months of pregnancy was associated with the preterm births. CONCLUSIONS: We found that there were consistent results when we applied the three different models. These findings suggest that air pollution exposure during the third trimester pregnancy has an adverse effect on preterm birth in South Korea.; Summary

Air Pollution Exposure and Health Effects in Fetus.: Bo Eun Lee, Hye sook Park, Young Ju Kim, Eun Ae Park, Yun Chul Hong, Eun Hee Ha; J Prev Med Public Health. 2004;37(4):291-299. Published online November 30, 2004

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Abstract PDF: As there have been growing concerns about the adverse effects of air pollution on birth outcome, studies for this area has been carried out in different populations and sites. We reviewed the epidemiologic studies that evaluated the effects of air pollution on birth outcome such as low birth weight and preterm births. We identified the air pollution exposure during pregnancy was related with low birth weight and preterm birth, although there are differences among studies for the critical period of vulnerability. The biological mechanisms whereby air pollution might influence health of fetus are not clearly established. The exposure to carbon monoxide (CO) during pregnancy could increase fetal carboxyhemoglobin and result in tissue hypoxia. On the other hand, ambient particles less than 10 micrometer in aerodynamic diameter (PM10) could lead to inflammation and increase blood viscosity. Controlling for potential confounders and valid assessment of exposure are the methodological issues remained in these epidemiologic studies. In the future, more studies are needed to investigate the effect of air pollution on preterm birth or stillbirths, considering the various exposure period and the biological mechanism.; Summary

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